Morphology Monday | Case MM250804
This week’s case comes from a 69 year old male presenting with a increasing fatigue and back back over the past several months. He has also experiences recurrent infections recently.
MM250721: Acute lymphoblastic leukaemia (ALL)
The findings are highly suggestive of an acute leukaemia, and given the age of the patient it raises a strong suspicion for acute lymphoblastic leukaemia (ALL)
Morphology Monday | Case MM250721
This week’s case features a 14-year-old male who had just celebrated his birthday when he presented to hospital with an abnormal full blood count and some concerning symptoms.
MM250707: Splenic marginal zone lymphoma (SMZL)
This week’s case featured a patient with Splenic Marginal Zone Lymphoma (SMZL) a rare, indolent B-cell lymphoma typically involving the spleen, bone marrow, and peripheral blood
Morphology Monday | Case MM250707
…a patient who presented with fatigue and splenomegaly on examination. Routine bloods showed a significant lymphocytosis, prompting further investigation.
MM250623: Acute Myeloid Leukaemia
The findings are strongly suggestive of acute leukaemia of myeloid origin, though further flow cytometry and molecular testing are essential for classification.
Morphology Monday | Case MM250623
a patient who presented with a gastrointestinal (GI) bleed and was started on tranexamic acid (TXA) as part of their management.
MM250616: May Hegglin Anomaly
This week’s case focused on an adult male with a platelet count in the 54×0⁹/L, identified during routine testing. The patient was asymptomatic, with no significant bleeding history, and haemoglobin and white cell counts were within normal limits. A blood film was reviewed as part of the thrombocytopaenia workup and revealed characteristic findings consistent with…
Morphology Monday | Case MM250616
Today’s case involves an adult male whose blood film was reviewed following an unexpected finding on a routine full blood count.
MM250609: Chronic myeloid leukaemia (CML)
CML is a myeloproliferative neoplasm driven by the BCR:ABL1 fusion gene